Marjana Tomic-Canic

A gene signature of nonhealing venous ulcers: potential diagnostic markers.

Retinoid-responsive transcriptional changes in epidermal keratinocytes.

Operative debridement of pressure ulcers.

Farnesyl pyrophosphate inhibits epithelialization and wound healing through the glucocorticoid receptor.

Attenuation of the transforming growth factor beta-signaling pathway in chronic venous ulcers.

The role of surgical debridement in healing of diabetic foot ulcers.

Hair cycling and wound healing: to pluck or not to pluck?

Cortisol synthesis in epidermis is induced by IL-1 and tissue injury.

The role of surgical debridement in healing of diabetic foot ulcers.

Keratin dressings speed epithelialization of deep partial-thickness wounds.

Micro-RNAs: New Regulators of Wound Healing.

Induction of specific microRNAs inhibits cutaneous wound healing.

Interactions of methicillin resistant Staphylococcus aureus USA300 and Pseudomonas aeruginosa in polymicrobial wound infection.

Quality assessment of tissue specimens for studies of diabetic foot ulcers.

Glucocorticoid receptor localizes to adherens junctions at the plasma membrane of keratinocytes.

Deep tissue injury in development of pressure ulcers: a decrease of inflammasome activation and changes in human skin morphology in response to aging and mechanical load.

Increased number of Langerhans cells in the epidermis of diabetic foot ulcers correlates with healing outcome.

Stress-Induced Hormones Cortisol and Epinephrine Impair Wound Epithelization.

Deregulation of epidermal stem cell niche contributes to pathogenesis of nonhealing venous ulcers.

Epidermal Differentiation in Barrier Maintenance and Wound Healing.

Wound research funding from alternative sources of federal funds in 2012.

Clinical application of growth factors and cytokines in wound healing.

Epithelialization in Wound Healing: A Comprehensive Review.

Stressing the steroids in skin: paradox or fine-tuning?

Wound repair and regeneration: mechanisms, signaling, and translation.

Effect of physical therapy on wound healing and quality of life in patients with venous leg ulcers: a systematic review.

Skin under the (Spot)-Light: Cross-Talk with the Central Hypothalamic-Pituitary-Adrenal (HPA) Axis.

Nanoparticles for Fidgety Cell Movement and Enhanced Wound Healing.

Comparative Genomic, MicroRNA, and Tissue Analyses Reveal Subtle Differences between Non-Diabetic and Diabetic Foot Skin.

Skin Metabolite, Farnesyl Pyrophosphate, Regulates Epidermal Response to Inflammation, Oxidative Stress, and Migration.

Epithelial-mesenchymal transition in tissue repair and fibrosis.

Biology and Biomarkers for Wound Healing.

Modalities to Treat Venous Ulcers: Compression, Surgery, and Bioengineered Tissue.

Integrative analysis of miRNA and mRNA paired expression profiling of primary fibroblast derived from diabetic foot ulcers reveals multiple impaired cellular functions.

Mineralocorticoid Receptor Antagonists-A New Sprinkle of Salt and Youth.

MiR-21 and miR-205 are induced in invasive cutaneous squamous cell carcinomas.

Stress Signals, Mediated by Membranous Glucocorticoid Receptor, Activate PLC/PKC/GSK-3β/β-catenin Pathway to Inhibit Wound Closure.

A bioengineered living cell construct activates an acute wound healing response in venous leg ulcers.

Updates in wound healing: Mechanisms and translation.

Wound healing protects against chemotherapy-induced alopecia in young rats via up-regulating interleukin-1β-mediated signaling.

Topical mevastatin promotes wound healing by inhibiting the transcription factor c-Myc via the glucocorticoid receptor and the long non-coding RNA Gas5.

Optical coherence tomography for assessment of epithelialization in a human ex vivo wound model.

Staphylococcus aureus Triggers Induction of miR-15B-5P to Diminish DNA Repair and Deregulate Inflammatory Response in Diabetic Foot Ulcers.

Probiotics or pro-healers: the role of beneficial bacteria in tissue repair.

A Modeling Conundrum: Murine Models for Cutaneous Wound Healing.

Descriptive vs mechanistic scientific approach to study wound healing and its inhibition: Is there a value of translational research involving human subjects?

Novel mevalonate kinase missense mutation in a patient with disseminated superficial actinic porokeratosis.

Differentiation of diabetic foot ulcer-derived induced pluripotent stem cells reveals distinct cellular and tissue phenotypes.

Increased IL-17-expressing γδ T cells in seborrhoeic dermatitis-like lesions of the Mpzl3 knockout mice.

Single cell analyses reveal specific distribution of anti-bacterial molecule Perforin-2 in human skin and its modulation by wounding and Staphylococcus aureus infection.

A tractable, simplified ex vivo human skin model of wound infection.

Cellular Senescence in Diabetic Wounds: When Too Many Retirees Stress the System.

Healing Chronic Wounds: Current Challenges and Potential Solutions.

Pharmacological and Genetic Inhibition of Caveolin-1 Promotes Epithelialization and Wound Closure.

Mevastatin promotes healing by targeting caveolin-1 to restore EGFR signaling.

A bioengineered living cell construct activates metallothionein/zinc/MMP8 and inhibits TGFβ to stimulate remodeling of fibrotic venous leg ulcers.

Multimodal, in Situ Imaging of Ex Vivo Human Skin Reveals Decrease of Cholesterol Sulfate in the Neoepithelium during Acute Wound Healing.

The Potential Impact of Social Genomics on Wound Healing.

Topical L-thyroxine: The Cinderella among hormones waiting to dance on the floor of dermatological therapy?

Skin Microbiota and its Interplay with Wound Healing.

Deregulated immune cell recruitment orchestrated by FOXM1 impairs human diabetic wound healing.

Advances in Stem Cell-Based Therapy for Hair Loss.

Boosts Innate Immune Response by Activation of Gamma Delta T Cells and Induction of Perforin-2 in Human Skin.

Intracellular escape strategies of Staphylococcus aureus in persistent cutaneous infections.

Genomics of Human Fibrotic Diseases: Disordered Wound Healing Response.

Epigenetic regulation of cellular functions in wound healing.

Cellular reprogramming of diabetic foot ulcer fibroblasts triggers pro-healing miRNA-mediated epigenetic signature.

Advanced Wound Diagnostics: Toward Transforming Wound Care into Precision Medicine.

Glucocorticoid-mediated induction of caveolin-1 disrupts cytoskeletal organization, inhibits cell migration and re-epithelialization of non-healing wounds.

Corrigendum: Boosts Innate Immune Response by Activation of Gamma Delta T Cells and Induction of Perforin-2 in Human Skin.

Intracellular Staphylococcus aureus triggers pyroptosis and contributes to inhibition of healing due to perforin-2 suppression.

Dichotomous role of miR193b-3p in diabetic foot ulcers maintains inhibition of healing and suppression of tumor formation.

FOXM1 network in association with TREM1 suppression regulates NET formation in diabetic foot ulcers.

Integrative small and long RNA omics analysis of human healing and nonhealing wounds discovers cooperating microRNAs as therapeutic targets.

Innate immunity and microbial dysbiosis in hidradenitis suppurativa - vicious cycle of chronic inflammation.

The NIDDK Diabetic Foot Consortium.

Molecular Pathophysiology of Chronic Wounds: Current State and Future Directions.

Urine-derived exosomes from individuals with IPF carry pro-fibrotic cargo.

Chronic wound microenvironment mediates selection of biofilm-forming multi drug resistant with capability to impair healing.

Novel Diagnostic Technologies and Therapeutic Approaches Targeting Chronic Wound Biofilms and Microbiota.

Cutaneous changes in diabetic patients: Primed for aberrant healing?

Calreticulin: a multifunctional protein with potential therapeutic applications for chronic wounds.

An Optimized and Advanced Algorithm for the Quantification of Immunohistochemical Biomarkers in Keratinocytes.

Postbiotic lactobacilli induce cutaneous antimicrobial response and restore the barrier to inhibit the intracellular invasion of Staphylococcus aureus in vitro and ex vivo.

Flipping the Script: Are Cellular Assays and Wound Fluids the Next Frontier in Personalized Wound Care?

Multi-drug resistant Staphylococcus epidermidis from chronic wounds impair healing in human wound model.

The Wound Reporting in Animal and Human Preclinical Studies (WRAHPS) Guidelines.

Collaboration encourages innovation: setting new standards in wound care with the Wound Care Collaborative Community expert panel consensus Recommendations.